We discuss a test of the CDF dijet anomaly at the LHC. The recent observed dijet mass peak at the CDF is well fitted by a new particle with a mass of around 150 GeV, which decays into two jets. In ...this paper, we focus on only \(Wjj\) signal to avoid model dependence, and comprehensively study the LHC discovery/exclusion reach. We found almost all the models are inconsistent with the result of the LHC, unless only valence quarks contribute the new process. We also discuss further prospects of the LHC search for this anomaly.
In this paper, we consider phenomenology of a model with an \(L_\mu-L_\tau\) gauge symmetry. Since the muon couples to the \(L_\mu-L_\tau\) gauge boson (called \(Z''\) boson), its contribution to the ...muon anomalous magnetic moment (muon g-2) can account for the discrepancy between the standard model prediction and the experimental measurements. On the other hand, the \(Z''\) boson does not interact with the electron and quarks, and hence there are no strong constraints from collider experiments even if the \(Z''\) boson mass is of the order of the electroweak scale. We show an allowed region of a parameter space in the \(L_\mu-L_\tau\) symmetric model, taking into account consistency with the electroweak precision measurements as well as the muon g-2. We study the Large Hadron Collider (LHC) phenomenology, and show that the current and future data would probe the interesting parameter space for this model.
To explore the biologic significance of the membrane-anchored form of macrophage colony-stimulating factor (M-CSF), we examined whether interaction between membrane-bound M-CSF and its receptor ...mediates intercellular adhesion as well as cell proliferation and differentiation. Human M-CSF receptors were expressed on a murine interleukin-2 (IL-3)-dependent cell line, FDC-P2, by DNA transfection with the c-fms gene (FDC-P2-MCSFR). A human bone marrow-derived stromal cell line, KM102, was used in the cell adhesion assay. The expression of membrane-bound M-CSF on KM102 cells was demonstrated by flow cytometry and immunoblot analysis. After the incubation of parent and transformed FDC-P2 cells on confluent KM102 cells, nonadherent cells were removed and the cells attached to KM102 cells were examined microscopically. Almost all parent FDC-P2 cells were nonadherent, whereas a significant number of FDC-P2-MCSFR cells bound to KM102 cells. The addition of anti-M-CSF or anti-M-CSF receptor neutralizing antibodies dose-dependently inhibited the binding of 3H-thymidine-labeled FDC-P2-MCSFR cells to KM102 cells. An excess amount of M-CSF also inhibited the binding. On the other hand, the addition of antibodies against some representative adhesion molecules (vitronectin receptor, Pgp-1/CD44, and VLA-4) did not inhibit the adhesion between FDC-P2-MCSFR cells and KM102 cells. These results support the idea that membrane-anchored M-CSF and its receptor may function as mediators of cell-cell adhesion.
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