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Peluso, Michael J; Mitchell, Anthea; Wang, Chung Yu; Takahashi, Saki; Hoh, Rebecca; Tai, Viva; Durstenfeld, Matthew S; Hsue, Priscilla Y; Kelly, J Daniel; Martin, Jeffrey N; Wilson, Michael R; Greenhouse, Bryan; Deeks, Steven G; DeRisi, Joseph L; Henrich, Timothy J
The Journal of infectious diseases, 01/2023, Letnik: 227, Številka: 2Journal Article
Abstract Interferon (IFN)–specific autoantibodies have been implicated in severe coronavirus disease 2019 (COVID-19) and have been proposed as a potential driver of the persistent symptoms characterizing “long COVID,” a type of postacute sequelae of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection. We report that only 2 of 215 participants with convalescent SARS-CoV-2 infection tested over 394 time points, including 121 people experiencing long COVID symptoms, had detectable IFN-α2 antibodies. Both had been hospitalized during the acute phase of the infection. These data suggest that persistent anti-IFN antibodies, although a potential driver of severe COVID-19, are unlikely to contribute to long COVID symptoms in the postacute phase of the infection. Within a cohort of individuals with well-characterized post–coronavirus disease 2019 (COVID-19) conditions, or “long COVID,” the authors identified only 2 with anti–interferon α2 autoantibodies, suggesting that these antibodies are unlikely to contribute to most cases of long COVID.
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JCR | SNIP | JCR | SNIP | JCR | SNIP | JCR | SNIP |
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Vir: Osebne bibliografije
in: SICRIS
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