We report new measurements of the production cross sections of pairs of charged pions and kaons as a function of their fractional energies using various fractional-energy definitions. Two different ...fractional-energy definitions were used and compared to the conventional fractional-energy definition reported previously. The new variables aim at either identifying dihadron cross sections in terms of single-hadron fragmentation functions, or to provide a means of characterizing the transverse momentum created in the fragmentation process. The results were obtained applying the updated initial-state radiation correction used in other recent Belle publications on light-hadron production cross sections. In addition, production cross sections of single charged pions, kaons, and protons were also updated using this initial-state radiation correction. The cross sections are obtained from a 558 fb-1 data sample collected at the Υ(4S) resonance with the Belle detector at the KEKB asymmetric-energy e+e- collider.
Temporal processing is traditionally dissociated from spatial vision. Recent evidence, however, has shown that adaptation to high temporal frequency (D. Burr, A.Tozzi, & M. C. Morrone, 2007; A. ...Johnston, D. H. Arnold, & S. Nishida, 2006; A. Johnston et al., 2008) induces spatially specific reductions in the apparent duration of subsecond intervals containing medium frequency drift or flicker. Here we examine the spatial tuning of these temporal adaptation effects. Our results show that duration compression is tightly tuned to the spatial location of the adaptor and can be induced by very narrow adaptors. We also demonstrate that the effects of adaptation on perceived duration are dissociable from those on apparent temporal frequency, which suggests early but separate influences of temporal frequency adaptation on time and speed perception.
We report the results of a high-statistics search for H dibaryon production in inclusive Υ(1S) and Υ(2S) decays. No indication of an H dibaryon with a mass near the M(H)=2m(Λ) threshold is seen in ...either the H→Λpπ(-) or ΛΛ decay channels and 90% confidence level branching-fraction upper limits are set that are between one and two orders of magnitude below the measured branching fractions for inclusive Υ(1S) and Υ(2S) decays to antideuterons. Since Υ(1S,2S) decays produce flavor-SU(3)-symmetric final states, these results put stringent constraints on H dibaryon properties. The results are based on analyses of 102 million Υ(1S) and 158 million Υ(2S) events collected with the Belle detector at the KEKB e(+)e(-) collider.
The Wilms' tumor gene WT1 is overexpressed in leukemia and solid tumors and has an oncogenic role in leukemogenesis and tumorigenesis. However, precise regulatory mechanisms of WT1 overexpression ...remain undetermined. In the present study, microRNA-125a (miR-125a) was identified as a miRNA that suppressed WT1 expression via binding to the WT1-3'UTR. MiR-125a knockout mice overexpressed WT1, developed myeloproliferative disorder (MPD) characterized by expansion of myeloid cells in bone marrow (BM), spleen and peripheral blood, and displayed urogenital abnormalities. Silencing of WT1 expression in hematopoietic stem/progenitor cells of miR-125a knockout MPD mice by short-hairpin RNA inhibited myeloid colony formation in vitro. Furthermore, the incidence and severity of MPD were lower in miR-125a (-/-) mice than in miR-125a (+/-) mice, indicating the operation of compensatory mechanisms for the complete loss of miR-125a. To elucidate the compensatory mechanisms, miRNA array was performed. MiR-486 was occasionally induced in compete loss of miR-125a and inhibited WT1 expression instead of miR-125a, resulting in the cancellation of MPD occurrence. These results showed for the first time the post-transcriptional regulatory mechanisms of WT1 by both miR-125a and miR-486 and should contribute to the elucidation of mechanisms of normal hematopoiesis and kidney development.
We present the first model-independent measurement of the CKM unitarity triangle angle ϕ3 using B±→ D(KS0\ {K}_{\mathrm{S}}^0 \π+π−π0) K± decays, where D indicates either a D0 or D¯\ \overline{D} \0 ...meson. Measurements of the strong-phase difference of the D →KS0\ {K}_{\mathrm{S}}^0 \π+π−π0 amplitude obtained from CLEO-c data are used as input. This analysis is based on the full Belle data set of 772 × 106BB¯\ \overline{B} \ events collected at the Υ(4S) resonance. We obtain ϕ3 = (5.7−8.8+10.2\ {5.7}_{-8.8}^{+10.2} \±3.5±5.7)° and the suppressed amplitude ratio rB = 0.323±0.147±0.023±0.051. Here the first uncertainty is statistical, the second is the experimental systematic, and the third is due to the precision of the strong-phase parameters measured from CLEO-c data. The 95% confidence interval on ϕ3 is (−29.7, 109.5)°, which is consistent with the current world average.
We report the first observation of e;{+}e;{-}-->Upsilon(1S)pi;{+}pi;{-}, Upsilon(2S)pi;{+}pi;{-}, and first evidence for e;{+}e;{-}-->Upsilon(3S)pi;{+}pi;{-}, Upsilon(1S)K+K-, near the peak of the ...Upsilon(5S) resonance at sqrts approximately 10.87 GeV. The results are based on a data sample of 21.7 fb;{-1} collected with the Belle detector at the KEKB e;{+}e;{-} collider. Attributing the signals to the Upsilon(5S) resonance, the partial widths Gamma(Upsilon(5S)-->Upsilon(1S)pi;{+}pi;{-})=0.59+/-0.04(stat)+/-0.09(syst) MeV and Gamma(Upsilon(5S)-->Upsilon(2S)pi;{+}pi;{-})=0.85+/-0.07(stat)+/-0.16(syst) MeV are obtained from the observed cross sections. These values exceed by more than 2 orders of magnitude the previously measured partial widths for dipion transitions between lower Upsilon resonances.
The presence of elevated calculated panel reactive antibody (cPRA) and anti‐HLA donor specific antibodies (DSA) are high risk factors for acute antibody‐mediated rejection (AAMR) in intestinal ...transplantation that may lead to graft loss. Eculizumab has been used for the treatment of AAMR in kidney transplantation of sensitized patients that do not respond to other treatment. Here, we report a case where eculizumab was used to treat AAMR in a desensitization‐resistant intestinal re‐transplant patient. A male patient lost his intestinal graft to AAMR 8.14 years after his primary transplant. He received a second intestinal graft that had to be explanted a month later due to refractory AAMR. The patient remained highly sensitized despite multiple treatments. He received a multivisceral graft and presented with severe AAMR on day 3 posttransplantation. The AAMR was successfully treated with eculizumab. The patient presently maintains an elevated cPRA level above 90% but his DSAs have decreased from 18 000 MFI (mean fluorescent intensity) to below the positive cut‐off value of 3000 MFI and remains rejection free with a 2‐year follow‐up since his multivisceral transplant. Eculizumab offers an alternative to treat AAMR in intestinal transplantation in desensitization‐resistant patients.
The authors report a case in which eculizumab, a humanized monoclonal antibody against complement C5, was used to treat acute antibody‐mediated rejection in a desensitization‐resistant intestinal re‐transplant patient.
We search for lepton-flavor-violating τ→ℓV0 decays, where ℓ is an electron or muon and V0 is one of the vector mesons ρ0, ϕ, ω, K⁎0 and K¯⁎0. We use 854 fb−1 of data collected with the Belle detector ...at the KEKB asymmetric-energy e+e− collider. No evidence for a signal is found in any decay mode, and we obtain 90% confidence level upper limits on the individual branching fractions in the range (1.2–8.4)×10−8.
We report measurements of B→χ(c1)γK and χ(c2)γK decays using 772×10(6) BBover ¯ events collected at the Υ(4S) resonance with the Belle detector at the KEKB asymmetric-energy e(+)e(-) collider. ...Evidence of a new resonance in the χ(c1)γ final state is found with a statistical significance of 3.8σ. This state has a mass of 3823.1±1.8(stat)±0.7(syst) MeV/c(2), a value that is consistent with theoretical expectations for the previously unseen 1(3)D(2) ccover ¯ meson. We find no other narrow resonance and set upper limits on the branching fractions of the X(3872)→χ(c1)γ and χ(c2)γ decays.
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