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Rietschel, M; Krauss, H; Mueller, D J; Schulze, T G; Knapp, M; Marwinski, K; Maroldt, A-O; Paus, S; Gruenhage, F; Propping, P; Maier, W; Held, T; Nothen, M M
European archives of psychiatry and clinical neuroscience, 02/2000, Letnik: 250, Številka: 1Journal Article
In the search for genetic factors contributing to tardive dyskinesia, dopamine receptor genes are considered major candidates. The dopamine D sub(3) receptor is of primary interest as dopamine D sub(3) receptor knock-out mice show locomotor hyperactivation resembling extrapyramidal side-effects of neuroleptic treatment. Furthermore, Steen and colleagues (1997) recently reported an association between tardive dyskinesia and a dopamine D3 receptor gene variant. In the present study we tried to replicate this finding. We investigated 157 patients with schizophrenia or schizoaffective disorder receiving long-term neuroleptic medication who never or persistently displayed tardive dyskinesia. As advanced age is a main risk factor for tardive dyskinesia, we also compared older patients with a long duration of schizophrenia not displaying tardive dyskinesia to younger patients with a shorter duration of the illness displaying tardive dyskinesia. However, we found no evidence that the dopamine D sub(3) receptor gene is likely to confer susceptibility to the development of tardive dyskinesia.
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JCR | SNIP | JCR | SNIP | JCR | SNIP | JCR | SNIP |
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in: SICRIS
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