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Microbiological diagnosis of viral hepatitisPoljak, Mario, 1965- ...Viral hepatitis is mainly caused by five viruses: hepatitis A virus (HAV), hepatitis B virus (HBV), hepatitis C virus (HCV), hepatitis D virus (HDV), andhepatitis E virus (HEV). All five hepatitis ... viruses can cause acute hepatitis, whereas HBV, HCV and HDV can also cause chronic hepatitis. The presence of anti-HAV IgM antibodies confirms the diagnosis of acute hepatitis A. The presence of total anti-HAV antibodies, in the absence of anti-HAV IgM, indicates previous infection with HAV or successful immunization. Six serological HBV markers are routinely determined, namely two viral proteins (HBsAg and HBeAg) and four anti-HBV antibodies (anti-HBs antibodies, total anti-HBc antibodies, anti-HBc IgM antibodies and anti-HBe antibodies). The detection of HBV DNA has recently become indispensable for resolving diagnostic uncertainties following serological testing for HBV markers. The diagnosis of hepatitis C begins with the detection of anti-HCV antibodies. In an immunocompetent subject, a negative result of anti-HCV screening test excludes chronic HCV infection. Individuals with repeated reactivity of anti-HCV screening test are tested for the presence of HCV RNA in order to distinguish those who have resolved HCV infection (HCV RNA negative) from the patients with active/ongoing HCV infection (HCV RNA positive). Three serological markers of HDV infection are routinely determined, namely total antiHDV antibodies, anti-HDV IgM antibodies, and hepatitis D antigen. The detection of anti-HEV IgM usually indicates an acute or recent infection with HEV. The presence of anti-HEV IgG antibodies alone, in the absence of anti-HEVIgM antibodies, indicates the recovery from hepatitis E or late phase of acute hepatitis E. Due to a low positive predictive value of testing for hepatitis E, the results of serological tests in Slovenian individuals with nohistory of recent travel to hepatitis-E-endemic areas should be interpreted with caution.Vir: Viral hepatitis : proceedings (Str. 72-86)Vrsta gradiva - prispevek na konferenciLeto - 2004Jezik - angleškiCOBISS.SI-ID - 18616025
Avtor
Poljak, Mario, 1965- |
Seme, Katja, 1966- |
Meško Meglič, Karmen |
Fujs Komloš, Kristina |
Štamol, Tina |
Babič, Dunja
Teme
Hepatitis, Viral, Human |
Microbiology |
Hepatitis A |
Microbiology |
Hepatitis B |
Microbiology |
Hepatitis B Antibodies |
Hepatitis B Surface Antigens |
Hepatitis B Core Antigens |
Hepatitis B E Antigens |
Dna, Viral |
Hepatitis C |
Microbiology |
Hepatitis C Antibodies |
Rna, Viral |
Hepatitis C Antigens |
Hepatitis E |
Microbiology |
Igg |
Igm |
Hepatitis A |
Hepatitis B |
Hepatitis B, površinski antigeni |
Hepatitis B, protitelesa |
Hepatitis virusni, človeški |
DNA virusna |
Hepatitis B e, antigeni |
Hepatitis B, sredica, antigeni |
Hepatitis C |
Hepatitis C, antigeni |
Hepatitis C, protitelesa |
Hepatitis E |
IgG |
IgM |
RNA virusna
Vnos na polico
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Povezave do osebnih bibliografij avtorjev | Povezave do podatkov o raziskovalcih v sistemu SICRIS |
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Poljak, Mario, 1965- | 11622 |
Seme, Katja, 1966- | 14504 |
Meško Meglič, Karmen | 24280 |
Fujs Komloš, Kristina | 25986 |
Štamol, Tina | 25988 |
Babič, Dunja | 22461 |
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