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HIROTA, Tomomitsu; TAKAHASHI, Atsushi; MIYATAKE, Akihiko; ENOMOTO, Tadao; NISHIYAMA, Chiharu; NAKANO, Nobuhiro; MAEDA, Keiko; OKUMURA, Ko; OGAWA, Hideoki; IKEDA, Shigaku; NOGUCHI, Emiko; SAKAMOTO, Tohru; KUBO, Michiaki; HIZAWA, Nobuyuki; EBE, Koji; SAEKI, Hidehisa; SASAKI, Takashi; EBIHARA, Tamotsu; AMAGAI, Masayuki; TAKEUCHI, Satoshi; FURUE, Masutaka; NAKAMURA, Yusuke; TAMARI, Mayumi; TSUNODA, Tatsuhiko; TOMITA, Kaori; SAKASHITA, Masafumi; YAMADA, Takechiyo; FUJIEDA, Shigeharu; TANAKA, Shota; DOI, Satoru
Nature genetics, 11/2012, Letnik: 44, Številka: 11Journal Article
Atopic dermatitis is a common inflammatory skin disease caused by interaction of genetic and environmental factors. On the basis of data from a genome-wide association study (GWAS) and a validation study comprising a total of 3,328 subjects with atopic dermatitis and 14,992 controls in the Japanese population, we report here 8 new susceptibility loci: IL1RL1-IL18R1-IL18RAP (P(combined) = 8.36 × 10(-18)), the major histocompatibility complex (MHC) region (P = 8.38 × 10(-20)), OR10A3-NLRP10 (P = 1.54 × 10(-22)), GLB1 (P = 2.77 × 10(-16)), CCDC80 (P = 1.56 × 10(-19)), CARD11 (P = 7.83 × 10(-9)), ZNF365 (P = 5.85 × 10(-20)) and CYP24A1-PFDN4 (P = 1.65 × 10(-8)). We also replicated the associations of the FLG, C11orf30, TMEM232-SLC25A46, TNFRSF6B-ZGPAT, OVOL1, ACTL9 and KIF3A-IL13 loci that were previously reported in GWAS of European and Chinese individuals and a meta-analysis of GWAS for atopic dermatitis. These findings advance the understanding of the genetic basis of atopic dermatitis.
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