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Garzorz-Stark, Natalie; Eyerich, Kilian
Journal of investigative dermatology, 05/2019, Letnik: 139, Številka: 5Journal Article
Psoriasis is a T helper type 17–mediated immune disease. Initial triggers that lead to T helper type 17 production and inflammatory cell recruitment into skin are being delineated. Autoantigens that stimulate T helper type 17 cells are also being identified. A new and important piece of the puzzle indicates that keratinocytes not only amplify inflammation, but that they are essential for a full-blown IL-17–mediated psoriatic phenotype in mice.
