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Shimoda, Kazuya; Shide, Kotaro; Kameda, Takuro; Hidaka, Tomonori; Kubuki, Yoko; Kamiunten, Ayako; Sekine, Masaaki; Akizuki, Keiichi; Shimoda, Haruko; Yamaji, Takumi; Nakamura, Kenichi; Abe, Hiroo; Miike, Tadashi; Iwakiri, Hisayoshi; Tahara, Yoshihiro; Sueta, Mitsue; Yamamoto, Shojiro; Hasuike, Satoru; Nagata, Kenji; Kitanaka, Akira
Journal of Clinical and Experimental Hematopathology, 2015, Volume: 55, Issue: 3Journal Article
Loss-of-function of ten-eleven translocation-2 (TET2) is a common event in myeloid malignancies, and plays pleiotropic roles, including augmenting stem cell self-renewal and skewing hematopoietic cells to the myeloid lineage. TET2 mutation has also been reported in lymphoid malignancies; 5.7~12% of diffuse large B-cell lymphomas and 18~83% of angioimmunoblastic T-cell lymphomas had TET2 mutations. We investigated TET2 mutations in 22 adult T-cell leukemia/lymphoma (ATLL) patients and identified a missense mutation in 3 cases (14%). TET2 mutation occurred in a number of ATLL patients and was likely involved in their leukemogenesis. J Clin Exp Hematop 55(3) : 145-149, 2015
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