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Rhee, C K; Yoo, K H; Lee, J H; Park, M J; Kim, W J; Park, Y B; Hwang, Y I; Kim, Y S; Jung, J Y; Moon, J Y; Rhee, Y K; Park, H K; Lim, J H; Park, H Y; Lee, S W; Kim, Y H; Lee, S H; Yoon, H K; Kim, J W; Kim, J S; Kim, Y K; Oh, Y M; Lee, S D; Kim, H J
The international journal of tuberculosis and lung disease 17, Številka: 1Journal Article
Multicentre study. To define the clinical characteristics of patients with tuberculosis (TB) destroyed lung due to past TB. We reviewed patients with TB-destroyed lung between May 2005 and June 2011. A total of 595 patients from 21 hospitals were enrolled. The mean age was 65.63 ± 0.47 (mean ± standard error); 60.5% were male. The mean number of lobes involved was 2.59 ± 0.05. Pleural thickening was observed in 54.1% of the patients. Mean forced vital capacity (FVC), forced expiratory volume in 1 s (FEV(1)), FEV(1)/FVC, bronchodilator response and number of exacerbations per year were respectively 2.06 ± 0.03 l (61.26% ± 0.79), 1.16 ± 0.02 l (49.05% ± 0.84), 58.03% ± 0.70, 5.70% ± 0.34, and 0.40 ± 0.04. The number of lobes involved was significantly correlated with FVC and FEV(1), and with the number of exacerbations per year. Use of long-acting muscarinic antagonists or long-acting beta-2 agonists plus inhaled corticosteroids resulted in bronchodilatory effects. Multivariable regression analysis showed that age, initial FEV(1) (%) and number of exacerbations during follow-up were independent factors affecting change in FEV(1). Decreased lung function with exacerbation, and progressive decline of FEV(1) were observed in patients with TB-destroyed lung.
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JCR | SNIP | JCR | SNIP | JCR | SNIP | JCR | SNIP |
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